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CELLIVITY PEPTIDE LABS · CEL-P-MOT-001
MOTS-c
Lyophilised research-vial concept
For laboratory research only · Not intended for human or veterinary use.
THAILAND LAUNCH PRICE฿2,25015 mg · per vial · tax and delivery confirmed at enquiry
Received · batch documentation under reviewAvailability is confirmed for this exact format before any commitment.
MOTS-c is a short peptide encoded within mitochondrial 12S ribosomal RNA and studied as a possible signal between mitochondrial and cellular metabolism. Most findings are preclinical, making it a mechanism-led research topic rather than an established therapy.
Research focus
Mitochondrial-derived peptide biology and cellular stress adaptation.
Current evidence context
Preclinical findings do not establish human therapeutic efficacy. Human drug-exposure and long-term safety information remain insufficient for a consumer longevity conclusion.
Questions this record follows
How does MOTS-c signalling change under metabolic or cellular stress?
Which preclinical findings reproduce across models and laboratories?
What human exposure, pharmacology, immunogenicity and safety data are missing?
Follow the sources
Registry entries identify reference substances. Literature links describe research and do not certify this product or its batch.
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Exact presentation review
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SELECTED PRODUCTMOTS-cCEL-P-MOT-001 · response after manual review
MOTS-c: before you enquire
What exactly does the selected format mean?+
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CELLIVITY
MOTS-c
CEL-P-MOT-001 · 10 mg / 15 mg / 40 mg
MOTS-c is a short peptide encoded within mitochondrial 12S ribosomal RNA and studied as a possible signal between mitochondrial and cellular metabolism. Most findings are preclinical, making it a mechanism-led research topic rather than an established therapy.
Mitochondrial-derived peptide biology and cellular stress adaptation.
Other names
MOTS-c · mitochondrial open reading frame of the 12S rRNA-c
PubChem CID
146675088
CAS number
1627580-64-6
Molecular formula
C101H152N28O22S2
Molecular weight
2174.6 g/mol
Peptide length
16 amino acids
Compound class
Mitochondrial-derived peptide
Follow the sources
Preclinical findings do not establish human therapeutic efficacy. Human drug-exposure and long-term safety information remain insufficient for a consumer longevity conclusion.
How does MOTS-c signalling change under metabolic or cellular stress?
Which preclinical findings reproduce across models and laboratories?
What human exposure, pharmacology, immunogenicity and safety data are missing?