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Semax is a synthetic heptapeptide derived from an ACTH fragment and studied in neurobiology. It has a region-specific clinical history, but evidence standards, authorised status and product formulations vary by market.
Research focus
ACTH-fragment-derived peptide research in neurobiology models.
Current evidence context
The global evidence base is limited and regionally concentrated. It does not establish a broadly approved cognitive-enhancement or longevity intervention, and a catalogue vial is not equivalent to any regional medicine.
Questions this record follows
Which proposed signalling effects reproduce in independent neurobiology models?
How do formulation and route change exposure and measured outcomes?
What larger, independently replicated human studies are still needed?
Follow the sources
Registry entries identify reference substances. Literature links describe research and do not certify this product or its batch.
Exact CELLIVITY-batch review is pending. Reference files below describe their own declared samples.
Purity, identity, per-vial content and sterility are separate results.
Product
Semax
Record
CEL-P-SMX-001
Files received
0
0 FILES
No product-specific laboratory file has been uploaded yet.
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SELECTED PRODUCTSemaxCEL-P-SMX-001 · response after manual review
Semax: before you enquire
What exactly does the selected format mean?+
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CELLIVITY
Semax
CEL-P-SMX-001 · 10 mg
Semax is a synthetic heptapeptide derived from an ACTH fragment and studied in neurobiology. It has a region-specific clinical history, but evidence standards, authorised status and product formulations vary by market.
ACTH-fragment-derived peptide research in neurobiology models.
Other names
ACTH(4-7)-Pro-Gly-Pro · MEHFPGP · Semax
PubChem CID
9811102
CAS number
80714-61-0
Molecular formula
C37H51N9O10S
Molecular weight
813.9 g/mol
Peptide length
7 amino acids
Compound class
Synthetic ACTH-fragment-derived heptapeptide
Follow the sources
The global evidence base is limited and regionally concentrated. It does not establish a broadly approved cognitive-enhancement or longevity intervention, and a catalogue vial is not equivalent to any regional medicine.
Which proposed signalling effects reproduce in independent neurobiology models?
How do formulation and route change exposure and measured outcomes?
What larger, independently replicated human studies are still needed?
Lab documents
CELLIVITY lot review pending
No lab files received
Regional use or small studies do not establish broad cognitive, neurological or longevity benefit.